Bøger i Biomathematical and Biomechanical Modeling of the Circulatory and Ventilatory Systems serien

This volume presents one of the clinical foundations of vasculopathies: the biological markers and risk factors associated with cardiovascular disease.

This book reviews signaling pathways in the regulation of circulatory and respiratory function, integrating biology, chemistry, and physics for a multidisciplinary view of physiological flows. It describes ion and molecular carriers and receptors.

Volumes 1 and 2 of Circulatory and Ventilatory Systems focus on cell structure and function (Volume 1) as well as cellular events that allow adaptation to environmental conditions, including cell proliferation and growth, migration, and death (Volume 2). Circadian cycles that drive rhythmic gene transcription are also covered.

Volume I of Circulatory and Ventilatory Systems focuses on cell structure and function (Part A) as well as cellular events that allow adaptation to environmental conditions (Part B).

This multidisciplinary review of signaling pathways that regulate circulatory and respiratory function focuses on those involved in cell signaling, from sensors and receptors on the cell surface to intracellular effectors that trigger molecule synthesis.

Together, the volumes in this series present all of the data needed at various length scales for a multidisciplinary approach to modeling and simulation of flows in the cardiovascular and ventilatory systems, especially multiscale modeling and coupled simulations.

The fifth volume in a series on modeling and simulation of flows in the cardiovascular and ventilatory systems, this book covers cells, tissues and organs of the cardiovascular and ventilatory systems, emphasizing mechanotransduction-based regulation of flow.

Providing physicians with vital support in generating computer models and simulations of flows in the cardiovascular and ventilatory systems, this book reviews the systems' anatomy and physiology and shows how to use signals and images as input data.